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These findings suggest that ascites-mediated ferroptosis resistance occurs through a mechanism distinct from -tocopherol transfer and establishes a unique microenvironmental context in which lipid remodeling is the dominant protective pathway
We hypothesize that GSSG produced by glutathione peroxidases in cytosol is transported into apoplastic spaces where class III peroxidases are located and serve as an oxidant to class III peroxidases during the production of ROS for plant defense
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