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Studies revealed that many COVID-19 patients had severely depleted glutathione stores, potentially contributing to the oxidative damage seen in serious cases
The results indicated lower risks of MACEs (14.8% vs
Inhibition of Sirt2 by AGK2 restricted both drug-sensitive and drug-resistant M.tb strains, enhanced the efficacy of isoniazid, reduced the bacillary burden and improved disease pathology in M.tb -infected mice [141]
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