A role for metalloendopeptidases in the breakdown of the gut hormone, PYY 336
Alterations in mitochondria-associated ER membranes, excessive production of reactive oxygen species, impaired autophagic flux, disturbed calcium transport, mitochondrial fragmentation, and activation of inflammatory pathways collectively drive progressive organellar dysfunction ( Within this framework, further consideration of AP through the lens of inter-organellar communication may offer an important conceptual advance
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Results from these trials, expected over the coming years, will provide more definitive evidence about efficacy and safety for cognitive outcomes
