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First, the functional outcome of METTL3 depends on the type of m6A reader protein it recruitsbinding with IGF2BP1 tends to stabilize SLC7A11 and inhibit ferroptosis (Liu et al., 2022), whereas interaction with YTHDC1 promotes ACSL4 expression and induces ferroptosis (Wu et al., 2024)

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Hence, it is difficult to see the need for a new framework for 21 st century medicine (171)