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Extensive epidermolysis and necrosis lead to thermoregulation disorder, fluid loss, and blood volume shortage

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MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis A study examining the peptide's expression in relation to exercise and skeletal-muscle measures across cell and rodent models

This study is to explore a GSH-responsive AP probe-net, which can be activated by the abundant GSH in ATC cells to generate reactive alkoxyamine, and the formed alkoxyamine can trap AP sites and block the APE1 activity, leading to ATC cell death but less adverse effects towards normal cells

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