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Zinc deficiency impairs neutrophil chemotaxis, as shown in models of crush syndrome where zinc chelation reduced neutrophil infiltration and muscle injury, indicating that adequate zinc levels are crucial for optimal neutrophil function migration [137]

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In those preclinical models it has been reported to promote angiogenesis the growth of new blood vessels through the VEGFR2AkteNOS / nitric-oxide pathway, which may help explain its observed effects on healing [1] [4]

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