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No inhibition of influx by the selective GLP-1 receptor antagonist exendin(939), suggesting that the GLP-1 receptor is not involved in the rapid entry into brain

This is followed by the release of nesfatin-1 which stimulates cholecystokinin secretion leading to a rise in bile salts from gallbladder emptying, and the binding of Takeda P protein coupled receptor 5 (TGR5) onto the surface of the enteroendocrine cells, all of which eventually culminates in GLP-1 secretion [9]

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