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co-administration with BPC-157 and TB-500 provides a favorable microenvironment for re-epithelialization, angiogenesis, and controlled remodeling, with potential experimental relevance in ulcers and hard-to-heal wounds

The table below represents a clinical summary of the two therapies across key categories: Primary mechanism: GHK-Cu acts as a tissue repair signal

A trained professional places a small needle in your arm to start the infusion

Thus the rapid reimport hypothesis explains both sets of data