doi: 10.3389/fphar.2023.1267765 Received 27 July 2023 Accepted 09 October 2023 Published 13 November 2023 Volume 14 - 2023 Edited by Xinwang Yang, Kunming Medical University, China Reviewed by Yipeng Wang, Soochow University, China Lin Wei, Anhui Medical University, China Updates Copyright 2023 He, Wang and Qu
Archiv Psychiatr Nervenkrankheit 1944;117:76136
We first describe the pathogenesis and pathophysiology of ARDS that involve dysregulated inflammation, alveolar-capillary barrier dysfunction, impaired alveolar fluid clearance and oxidative stress
For instance, when peripheral tissues become inflamed, the related signals might be transmitted to the brain through these receptors on the neurons, and the brain could then respond to these signals via neural pathways, thus regulating the level of peripheral inflammation
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