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Acknowledgements We acknowledge the DSFP contract, King Saud University, Riyadh, KSA (to SYA) and the Slovak Grant Agencies VEGA Project 1/0482/20 and APVV Project 19-0087

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The mechanism proposed involves chromatin remodeling at the hTERT promoter specifically, a reduction in the repressive histone modifications (H3K9me3, H3K27me3) that maintain transcriptional silencing in somatic cells allowing activating transcription factors to access promoter binding sites and initiate hTERT transcription

doi:10.1007/s11064-020-03030-1

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