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The mechanism likely involves both the direct mechanical benefit of reduced joint loading (less body weight pressing on the knee) and possible anti-inflammatory effects mediated through reductions in systemic inflammation a known consequence of significant weight loss and potentially enhanced by glucagon receptor-mediated metabolic improvements

However, in rodent studies, some of these medications were associated with both benign and malignant thyroid C cell tumors

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This issue has been attributed to the mode of action of these antagonists: those blocking CAP-, proton-, and heat-induced TRPV1 activation result in hyperthermia, whereas those sparing proton-induced activation do not lead to hyperthermia 33,35

GLP-1 medications reduce inflammation through two pathways