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ROS not only directly damage endothelial cells but also inhibit eNOS activity, further reducing NO production and aggravating vasoconstriction

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In cirrhotic patients peripheral blood mononuclear cells and gastric cancer cell lines, palmitoylcarnitine (C16:0) has been shown to directly integrate into the mitochondrial membrane, inducing membrane potential collapse, ROS production, and cytochrome c release, ultimately activating caspase-mediated apoptosis [124, 125]

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