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Furthermore, overexpressing SLC25A40 in SLC25A39 knockout cells was sufficient to restore mitochondrial GSH levels, suggesting its redundant function in enabling mitochondrial GSH import alongside SLC25A39 (Fig

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Results for specific activity were divided by g/Hb for erythrocyte specimens

This combination is frequently used in research models to examine both localized and systemic signaling mechanisms and how they may interact within complex biological systems