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Bioavailability via non-injection routes is essentially zero for both

Mice expressing tau mutations such as P301L or P301S have been extensively used to investigate how tau aggregation leads to neuronal dysfunction, synaptic loss, and cognitive decline, recapitulating the tau pathology seen in AD patients 137

Baseline and periodic measurement of vitamins A, D, E, and K may be warranted in patients with documented malabsorption

Ferroptosis is strikingly driven by worsening redox imbalance, which generates and maintains high levels of hydroperoxy-phospholipids (7, 24)