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Consequently, we focused our analysis on eWAT and demonstrate that Retatrutide reverses this dysfunctional phenotype through coordinated changes in gene expression governing lipid turnover, mitochondrial biogenesis, and extracellular matrix remodeling

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Part II: The Pattern (Four Factors That Predict Risk) If this mechanism is correct, dysesthesia should correlate with factors that reduce cellular ATP availability or increase cellular energy demand