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The basis of the development of AITD - Hashimotos thyroiditis (HT), Graves disease (GD), Graves ophthalmopathy (GO) is the loss of immune tolerance to thyroid antigens - thyroid peroxidase (TPO), thyroglobulin (Tg) and thyroid-stimulating hormone receptor (TSH-R)

[1] Lower GSH levels are implicated in many diseases and GSH has been tried in the management of Alzheimers disease, Parkinsons disease, multiple sclerosis, alcoholic hepatitis, atherosclerosis, acquired immunodeficiency syndrome, and chronic fatigue syndrome

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