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Many of these genes contribute to homeostasis at the glutamate and GABA synapses, pointing to a potentially disrupted balance between glutamate and GABA transmission in the CNS- Glud1 +/ /Stress group
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Palazzuoli, V., Mondillo, S., Faglia, S., D'Aprile, N., Camporeale, A., and Gennari, C
These components are considered immunological distractors with pro-inflammatory activity, capable of causing dysbiosis and increased intestinal permeability (the so-called leaky gut syndrome), so favoring the development of autoimmune diseases [142]