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Molecular and cellular processes, such as myofibroblast/mesenchymal transition, myofibroblast activation and uncontrolled proliferation, endoplasmic reticulum stress, altered expression of growth factors, and oxidative stress, link PF to lung cancer and increase the risk of cancer development by 7%20% ( In addition, lung damage and fibrosis are frequently observed in tuberculosis (Ravimohan et al., 2018)

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