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GSTM3TV2
doi:10.1161/JAHA.114.001469
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Critically, it does not bind to the growth hormone receptor and does not raise IGF-1 levels.[1] This means its effects stay localized to fat metabolism, making it a much more targeted option than full hGH therapy
5 Conclusions Summarizing the above results, we speculate that glutathione can protect the liver of bullfrogs by repairing liver tissues, enhancing the antioxidant ability of bullfrogs, affecting the related metabolism of the body, repairing liver damage, up-regulating Autophagy - other, Necroptosis and ErbB signaling pathway, and It also up-regulated Autophagy - other, Necroptosis and ErbB signaling pathway, and down-regulated Sphingolipid metabolism and D-Amino acid metabolism pathway, and regulating the disorder of liver lipid metabolism
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