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One hypothesis is that we observed a greater induction of NRF2 through liver-specific Gclc deletion as compared to KEAP1 inhibition with CDDO-me because CDDO-me is a weaker inhibitor of NRF2-KEAP1 interaction compared to the ROS generated following GSH depletion

4 Potential role of ferroptosis mechanism in the treatment of epilepsy 4.1 Application of ferroptosis inhibitors and associated agents in the treatment of epilepsy Firstly, the exploration of the application of ferroptosis inhibitors in the treatment of epilepsy can be traced back to some fundamental research

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With the increased addition of GSH, the color of Glud-MFo-c changed (Figure 6A), suggesting that Fe 3 (CN) 6 was reduced to Fe 2 (CN) 6 and GSH was converted to GSSG (Figure 6B)

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