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Finally, the emergence and termination of some forms of plasticity during critical periods has been generally addressed in quiescent states out of considering a more physiological scenario where external inputs such as ongoing neuronal networks oscillations command the sign of plasticity as it may occurs in vivo

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Bcl-2 and OGC appear to act in a coordinated manner to increase the mGSH pool and to enhance the resistance of neurons to mitochondrial oxidative stress