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Its short half-life is a feature, not a limitation, preserving the physiologic pulsatility of GH secretion

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However, there are no high-quality human trials, no standardized dosing, and no long-term safety data

N1437D HOMO mutant mice harbor higher tau phosphorylation, lipofuscin accumulation, autophagy, and lysosomal abnormality in SN TH neurons at 2627 months of age Because N1437D mutations caused behavioral and histopathological damage at older ages, we selected mice aged 26 to 27 months to explore the molecular pathological features

doi: 10.1016/j.phrs.2017.09.010 227 DehzadMJGhalandariHNouriMAskarpourM