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Alcohol damages liver cells, and hence, more and more glutathione is utilized by our body to reduce this damage to the liver

It has been reported that oral administration of glutathione did not change the levels of glutathione and glutathione disulfide in the deproteinized fraction of blood [10], and it has been suggested that orally administered glutathione is degraded into constituting amino acids and does not exert specific activity beyond the amino acid source

The absence of PINK1 inhibits PINK1-dependent mitophagy, and although cells may clear mitochondria through other pathways, the efficiency of their elimination is reduced, resulting in the accumulation of damaged mitochondria, iron, and synuclein alpha (SNCA) or even ferroptosis in the substantia nigra of PD patients [176, 177]

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