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In vivo and in vitro experiments have shown that glutathione deficiency can lead to mitochondrial damage and cell death caused by an increase in the number of toxic forms of oxygen, leading to an increase in the number of free radicals

88 found that the activated PPAR could induce the expression of its target gene uncoupling protein 2 (UCP2) and increase the activity of fatty acid -oxidation-related enzymes and peroxidase in mitochondria

Protein-Derived Acetaminophen-Cysteine Can Be Detected After Repeated Supratherapeutic Ingestion of Acetaminophen in the Absence of Hepatotoxicity

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