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Association of insulin resistance with cerebral glucose uptake in late middle-aged adults at risk for Alzheimer disease

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These findings offer a new therapeutic strategy for this rare, aggressive malignancy and warrant further investigation in larger cohorts

One potential mechanism is the Nrf2-ARE pathway, which induces a phase 2 detoxification response that promotes disruption of Nrf2-Keap protein interactions, translocation of Nrf2 to the nucleus, and regulation of target gene expression through the ARE, enhancing cellular defense against oxidative damage (Sharma et al., 2010)

These include the hypothalamus, VTA, and nucleus accumbens