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Molecular targets for cancer therapy in the PI3K/AKT/mTOR pathway

For example, loss of SNX function could broadly disrupt endosomal membrane organization, phosphoinositide composition, or retromer recruitment, thereby indirectly impairing VSR recycling 20,51

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An early study showed that 250 M acetyl-l-carnitine did not affect axon regeneration but significantly reduced the rate of death of neurons from the DRG of aging rats, suggesting its neuroprotective action [14]