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Critically, both exercise modalities improve endothelial function through increased shear stress, which upregulates eNOS expression and NO bioavailability, thereby counteracting the endothelial dysfunction central to atherosclerosis (148)

[7] Vitamin D toxicity presents chiefly as sustained hypercalcaemia with nausea, vomiting, muscle weakness, neurocognitive changes, or arrhythmias, progressing to nephrocalcinosis if untreated

identified two genes, charged multivesicular body protein 1a (CHMP1A) and dipeptidase 1 (DPEP1), associated with kidney disease that could regulate ferroptosis by altering cellular iron transport [141]

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Gut inflammation in chronic fatigue syndrome