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Furthermore, these polymorphisms are associated with decreased MTHFR enzyme activity and hence alter the pool of 5,10-methyleneTHF substrate available for other biosynthetic pathways in the folate system ( i.e

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PGA is a degradation product of glutathione metabolism within the -glutamyl cycle and is directly associated with disruptions in GSH homeostasis and GSH depletion [44, 45, 65, 66]

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