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Damaged AECs release pro-inflammatory and pro-fibrotic mediators, including interleukin (IL) -1 and tumor necrosis factor (TNF) -, which promote fibroblast recruitment, inflammatory cell activation (e.g., neutrophils, Th17, gdT cells), and myofibroblast differentiation, ultimately leading to ECM accumulation and fibrotic tissue remodeling [20, 29, 33, 34]

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Conclusion IGF-1 LR3 is generating interest in aesthetic and anti-aging medicine thanks to its potential for muscle growth, fat metabolism, and skin repair

Metformin reduces hepatic glucose production and improves insulin sensitivity in muscle tissue, while GLP-1 drugs slow gastric emptying and enhance pancreatic insulin secretion

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