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Together, these interconnected signaling cascades reshape transcriptional programs in fibroblasts, AECs, and endothelial cells, promoting myofibroblast differentiation, epithelial cell dysfunction, immune dysregulation, and excessive ECM deposition, which collectively drive the onset and progression of pulmonary fibrosis [33, 39] (Fig

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Keay, S., Birder, L

Diallyl trisulfide induces Bcl2 and caspase3dependent apoptosis via downregulation of Akt phosphorylation in human T24 bladder cancer cells

Step 5: Verify the Solution The reconstituted solution should be completely clear with no visible particles, cloudiness, or discoloration