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1 Introduction Polycystic ovary syndrome (PCOS) represents one of the most complex endocrine-metabolic disorders affecting women of reproductive age, characterized by a collection of reproductive, metabolic, and psychological manifestations ( Beyond the classic triad of hyperandrogenism (HA), ovulatory dysfunction, and polycystic ovarian morphology, PCOS extends into domains of insulin resistance, dyslipidemia, chronic inflammation, and heightened oxidative stress (OS) ( Conventional therapeutic strategies, though effective in symptom control, remain largely palliative, targeting isolated components of this multifactorial syndrome ( Thus, this review critically examines the triadic potential of these agents, not as standalone supplements but as a synergistic bioactive cocktail targeting the redox-endocrine axis
49 , e81 (2021)
Using NMR metabolomics and flow cytometry to detect the Treg proportion, it was found that butyrate, as a histone deacetylase (HDAC) inhibitor, can enhance histone H3 acetylation of the Foxp3 gene and directly promote the differentiation of colonic Tregs
Hormonal Changes: Fluctuations in hormone levels, particularly during pregnancy, can trigger excess melanin production
