Thus, toxic metabolites of VPA can lead to an increase in the severity of neurological diseases and mental disorders [19], congenital malformations in the fetus [20], dysfunction of the neuroendocrine system [21], and impaired hematopoiesis [22]
Role of inflammation, immunity, and oxidative stress in hypertension: New insights and potential therapeutic targets
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GCG activation suppresses endogenous hepatic lipid production by inhibiting lipogenic enzymatic pathways while simultaneously enhancing mitochondrial oxidative lipid catabolism, thereby promoting lipid catabolism and reducing hepatic triglyceride content [18]
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